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In contrast with astrocytes, neurons, microglia, and oligodendrocytes do not readily release GSH (Hirrlinger et al., 2002c), and this is likely explained by the low level expression of MRP1 in these cell types (Hirrlinger et al., 2002a)

Indeed, OL loss in the cuprizone model can be rescued by treatment with a lipid radical scavenging molecule, ferrostatin 1 70

Cells were then double-stained with Annexin-V-PE and 7-amino-actinomycin D (7AAD) (PE Annexin V Apoptosis Detection Kit with 7-AAD

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reported that DZN induced oxidative stress in rat testes, indicated by increased lipid peroxidation and decreased GSH, Vitamin C and E, and -carotene levels, which could be mitigated by NAC treatment, which reduced lipid peroxidation and boosted antioxidant levels [41]

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