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Smoking-induced adipose dysfunction is characterized by altered adipocyte differentiation, impaired insulin action, and dysregulated adipokine secretion (274)

Fourth, DN involves multiple regulated cell-death pathways with potential crosstalk, and since apoptosis-related proteins (such as the caspase family) and autophagy markers (such as LC3 and p62) were not assessed, it cannot be excluded that DAPA also modulates these pathways and contributes to the observed protective effects

Activation of c-Jun-N-terminal kinase is required for apoptosis triggered by glutathione disulfide in neuroblastoma cells

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This switch supports more effective adenosine triphosphate (ATP) production, mitochondrial biogenesis, maturation, regulation of neurotransmitter release, maintenance of redox homeostasis, and resting membrane potential [1, 2]

2,82 Its photoprotective activities may guard against UV light-induced cell damage and cancer development, and its immunoenhancement may help to protect against cancer progression