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The authors confirmed that AngIV analogs can inhibit HGF binding to c-Met and HGF-dependent signaling by interfering with HGF dimerization, a crucial step for c-Met activation

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The ability of a quinone to redox cycle is dependent upon the favourability of its reduction by a single electron, which can be quantitatively described by its reduction potential ( E o )

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Apoptosis 11(6):889-904, 2006

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