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Mechanistically, plumbagin interferes with the YTHDF1-mediated m 6 A methylation pathway that regulates SLC7A5 translation (84)

and as the inflammatory response persists, more substantial cognitive deficits, such as impaired spatial memory formation, reduced cognitive flexibility, decreased processing speed, altered emotional regulation, and compromised learning ability, may emerge (Guan et al., 2009

Knockdown of YTH N(6)-methyladenosine RNA binding protein 2 (YTHDF2) inhibits proliferation and promotes apoptosis in MGC-803 gastric cancer cells]

In ferroptosis, SLC7A11 and GSH depletion lead to iron-dependent ROS accumulation ( Ferroptosis and alzheimers disease Iron homeostasis and alzheimers disease Regulation of iron homeostasis is important for maintaining normal brain function, and dysregulation of iron homeostasis in the brain can lead to oxidative stress and inflammatory responses, resulting in cell damage and ultimately neurodegenerative diseases

29 Early studies posited that single-stranded RNA could not form double-stranded structures