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British Journal of Clinical Pharmacology, 65 (1), 51-59

10.3389/fcell.2022.826204 126 ZhouR.LinJ.WuD

[DOI] [PubMed] [Google Scholar] 97.Koca R., Armutcu F., Altinyazar H.C., Grel A

To view a copy of this license, visit About this article Cite this article Meng, J., Fu, L., Liu, K

The activation is linked to the generation of vasoconstrictive agents, including MCP-1, (pro)renin receptor, endothelin, and angiotensin II, while concomitantly diminishing vasodilatory compounds like nitric oxide, which may contribute to the development of hypertension and lead to decreased viability of cardiomyocytes and myocardial damage