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Cyst(e)inase degrades extracellular cysteine and cystine, leading to reduced intracellular cysteine and glutathione levels, affecting cancer cells redox capacity (44, 140), inducing the accumulation of ROS (26) and consequent ferroptosis (135, 136)

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These are then converted into reactive oxidative mediators like hydrogen peroxide, constituting a primary pathway for cisplatin-induced ROS production [24, 25]

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