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Proinflammatory mediators in the bronchoalveolar lavage fluids from oxidant-expose lungs (CEES caused a 20-fold, fourfold, and 1.6-fold increase in CINC-1, IL-1, and TNF-, respectively) were reduced to baseline levels [99]

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Under stress conditions, ginsenoside Rb1 regulates the DUSP 1, TMBIM 6 and VDAC1 axis to regulate the mitochondrial function of cardiomyocytes, inhibit the inflammation development, and indirectly inhibit ferroptosis in cardiomyocytes 54

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