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Indeed, all neuroimaging studies that have compared ME/CFS patients with healthy controls demonstrated abnormalities in response to cognitive (Shan et al., 2016, 2017

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Among the molecular mechanisms proposed, glutamate excitotoxicity, dysregulation of transcription, altered RNA metabolism, defective axonal transport, protein misfolding and aggregation, endoplasmic reticulum stress, disrupted protein trafficking, proteasome impairment, inflammation, activation of cell death signals, mitochondrial dysfunction, and oxidative stress are the most discussed[2, 2932]

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