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Margiana, R

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4.3 Pathological changes in the liver caused by arsenic trioxide 4.3.1 Inflammatory mechanism Enhanced oxidative stress results in inflammation and the generation of pro-inflammatory cytokines, such as interleukin 6 (IL-6), interleukin 1 (IL-1), and tumor necrosis factor-alpha (TNF-), causing the disruption of the biological membrane ( 4.3.2 Fibrosis mechanism The networks of inflammation, cell death, and HSC activation mutually influence each other, resulting in initial hepatic steatosis and, with prolonged arsenic exposure, eventual portal fibrosis (Santra et al., 2000)

It supports gradual pigmentation reduction by regulating melanin production

This tumor, which was diagnosed as an anaplastic oligoastrocytoma (WHO grade III) that had progressed from a grade II tumor, exhibited FGFR1-TKDD positivity

Importantly, we also observed a significant reduction in OL differentiation following DMF and Clem-F application upon siRNA against Gsta4, which illustrates the importance of scavenging enzymes and cellular protection during differentiation