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Following injury, a complex inflammatory response is triggered

These single-cell expression profiles systematically delineate the spatiotemporal regulation patterns of ferroptosis-suppressing signature genes within the renal disease microenvironment, providing direct cellular and molecular evidence for elucidating the mechanisms of ferroptosis imbalance in AKD and CKD pathogenesis, while offering important references for subsequent research on intervention strategies targeting ferroptosis-suppressing pathways in kidney diseases

This may be caused by an inflammatory response in the tissue surrounding the spleen's blood vessels, which can exert pressure on the blood vessels and lead to splenic infarction [116]

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