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Matrine pretreatment improves cardiac function in rats with diabetic cardiomyopathy via suppressing ROS/TLR-4 signaling pathway

Following malignant transformation, ROS promote HCC progression by activating growth factors through the phosphatidylinositol 3-kinase (PI3K)/protein kinase B (AKT)/mammalian target of rapamycin (mTOR) and MAPK/extracellular signal-regulated kinase (ERK) mitogenic signaling cascades

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