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Description

Hyperglycemic states contribute to cerebral endothelial damage, promote atherosclerosis, affecting cerebral perfusion and function while impeding the clearance of brain metabolites

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The analysis of the subphenotype revealed that the treatment decreased the number of M2 macrophages that were identified as the profibrotic phenotype (Fig

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Discussion Aging manifests as a heterogeneous process among individuals and across distinct tissues within the same individual

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MCL1 is another antiapoptotic protein involved in leukemia cell survival

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G., Mohammadgholian, E., Amiri, M., Jafari, N., Rahimi, A., Alijani, E., & Earnest, C

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A recent study has further demonstrated that NIN administration markedly alleviated liver injury and fibrosis by significantly increasing autophagic activity in HSCs, concurrently inducing ferroptosis in mice subjected to BDL (Yu et al., 2024)

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